Can You Actually Reverse Type II Diabetes? The Truth Nobody Explains

Can type 2 diabetes actually be reversed? Well, the short answer is yes, for some people, not for everyone. And if anybody tells you that every case of diabetes can be cured with cinnamon, fasting, a supplement, or a one-stop type of special diet, they're not telling you the exact truth. If they are talking about parasite, definitely there's a parasite in their brain, not in their pancreas. Now, in this video, I am going to explain exactly what diabetes reversal really means, what is happening inside your liver, muscles, pancreas, and fat cells, who has the best chance of reaching remission, who may not be able to, and step-by-step strategy that will give you the greatest chance of success. I will also explain where medications, intermittent fasting, resistance training, sleep and supplements, including Berberine, SugarMD Super Berberine, Gluxion, all of that, how they may fit into that plan. This will be a comprehensive one, but it will not be complicated. So stay with me, because once you understand the physiology, diabetes becomes a lot easier to understand and reverse. I'm Dr. Ahmed Ergin. I'm an endocrinologist in Port St. Louis, Florida, owner of sugarmds.com, and I have spent my career treating diabetes, insulin resistance and obesity, thyroid disease, and hormonal disorders. So let's begin with the most important distinction. Reversal is not the same as a permanent cure. The preferred medical term is remission. According to the International Consensus definition, type 2 diabetes is considered in remission when your hemoglobin A1c remains below 6.5% for at least three months. Without glucose-lowering medication. That definition is important. If your A1c is 6.2% because you are taking metformin, insulin, semaglutide, terzepatide, or another diabetes medication, your diabetes may be beautifully controlled, but technically, you are not in remission. And there is nothing wrong with that. Excellent control with medication is much better than uncontrolled diabetes without medication. The goal is not to prove that you can live without medicine. The goal is to prevent blindness, kidney failure, neuropathy, heart attacks, strokes, amputations, and premature death. If remission is possible and safe, that is wonderful. If medication is still needed, that is not failure. Now let me explain what type 2 diabetes actually is. Imagine that insulin is a key. Its job is to help glucose move from your bloodstream into certain cells, especially muscle and fat cells. When insulin attaches to its receptor, it sends a message to the cell. Glucose is available, and the cell should respond. Early in the development of type 2 diabetes, insulin is usually not missing. In fact, many people with prediabetes initially produce too much insulin. The problem is that the body is no longer responding normally to it. This is insulin resistance. The key is present, but the lock has become difficult to turn. The pancreas responds by making more insulin. For years, this compensation may keep the glucose looking relatively normal. Your fasting glucose may be 95. Your A1c may be 5.6%. Your doctor may tell you everything looks fine. But underneath that normal glucose, your pancreas may be working overtime and producing two or three times as much insulin as it once did. This is why diabetes does not usually begin on the day your A1c reaches 6.5%. The disease process may have been developing silently for many years. Your insulin rises first. Then your glucose begins to rise after meals. Then your fasting glucose rises. Then your A1c enters the prediabetes range. And eventually, if the pancreas can no longer produce enough insulin to overcome the resistance, type 2 diabetes appears. So type 2 diabetes has two major components. Insulin resistance and insufficient insulin production relative to what your body requires. You may still produce insulin, but it is no longer enough to overcome the resistance. To understand reversal, we need to examine four major organs. The liver, the muscles, the fat tissue, and the pancreas. Let's begin with the liver. Your liver is a glucose warehouse. When you eat, it stores some glucose as glycogen. Between meals and overnight, it releases glucose to keep your brain and body functioning. Insulin normally tells the liver, we have enough glucose, stop releasing more. But when the liver becomes insulin resistant, it does not listen appropriately. The pancreas releases insulin. The message is sent. But the liver continues releasing glucose anyway. This is one of the main reasons fasting glucose becomes elevated. You can go to bed without eating anything and wake up with a glucose of 140. Patients often tell me, doctor, I did not eat anything. Where did the sugar come from? It came from your liver. Food is not the only source of blood glucose. Your liver can create glucose through a process called gluconeogenesis and release stored glucose through glycogenolysis. In type 2 diabetes, that glucose production is often excessive. Now let's look at skeletal muscle. Muscle is one of the largest destinations for glucose after a meal. When muscle is active and insulin sensitive, it acts like a large glucose reservoir. But when you lose muscle, remain inactive, or develop muscle insulin resistance, that reservoir becomes smaller and less responsive. Glucose remains in the bloodstream longer. The pancreas must release more insulin. And the metabolic problem becomes worse. This is why muscle is not just about looking fit. Muscle is part of your glucose control system. You cannot have a complete conversation about reversing type 2 diabetes without talking about building and preserving muscle. The third organ is body fat. Fat tissue is not simply a passive storage bag. It is hormonally active. Healthy fat tissue can store excess energy safely. But every person has a different capacity for safe fat storage. Once that capacity is exceeded, fat begins accumulating in places where it does not belong, particularly around the abdominal organs, inside the liver, inside muscle, and around the pancreas. This is called ectopic fat. You do not need to look severely obese to have ectopic fat. Some people develop type 2 diabetes at a relatively normal body weight, because their personal capacity to store fat safely is lower. Others can carry much more weight before their glucose rises. This is why diabetes cannot be diagnosed or understood by appearance alone. When excess fat accumulates inside the liver, liver insulin resistance worsens. The liver produces more glucose and may produce more triglyceride-rich particles. Fat can then be delivered to the pancreas and other tissues. Inside and around the pancreas, this abnormal fat environment can interfere with beta cell function. Beta cells are the cells that produce insulin. For years, the beta cells compensate by working harder. But eventually, susceptible beta cells begin to lose function. Some become exhausted. Some lose their normal identity. Some may die. And the pancreas can no longer keep up. Now the person has both insulin resistance and declining insulin secretion. That combination produces type 2 diabetes. But here is the hopeful part. At least some of this dysfunction can improve. When a person loses a meaningful amount of excess fat, liver fat can decrease. The liver may become more responsive to insulin. Excess glucose production may decline. Fat around the pancreas may decrease. In some people, particularly those earlier in the disease, the beta cells recover enough function to maintain normal glucose without medication. That is the biological basis of remission. You are not simply making glucose disappear. You are reducing the metabolic pressure that caused the glucose to rise. This is why substantial fat loss can be so powerful. In the direct trial, an intensive weight management program produced diabetes remission in almost half of participants after one year. The likelihood of remission was strongly related to the amount of weight lost. People who lost the most weight had the highest remission rates. The lesson is not that everyone must follow one specific liquid diet. The lesson is that meaningful and sustained loss of excess fat can fundamentally change the physiology of type 2 diabetes. But there is a second lesson. Remission can disappear if the weight returns. Type 2 diabetes has a memory. If you regain the visceral and ectopic fat that contributed to the disease, insulin resistance can return. Beta cell stress can return. And glucose can rise again. That is why I prefer the term remission rather than cure. The fire may be out, but the conditions that created the fire can return. Now, who has the best chance of reaching remission? The first group is people diagnosed relatively recently. If you were diagnosed 6 months ago, 2 years ago, or even several years ago, your beta cells may still have meaningful reserve. The earlier you intervene, the greater the opportunity to recover function before too much beta cell capacity is lost. The second group is people who still produce adequate insulin. We can sometimes estimate this with a C-peptide level. C-peptide is released when your pancreas produces insulin. A preserved C-peptide suggests that your pancreas still has insulin-producing capacity. But C-peptide must be interpreted with the glucose level, kidney function, timing, and whether the person is fasting. It is not a simple pass or fail test. The third group is people whose diabetes is strongly driven by excess visceral and ectopic fat. If weight gain was followed by worsening glucose, and meaningful fat loss produces rapid improvement, remission may be achievable. The fourth group is people who can lose enough weight and maintain that loss. For one person, losing 7% of body weight may produce major improvement. Another may need 10%, 15%, 20%, or more to reach remission. There is no universal magic number. Your body has its own personal fat threshold. The goal is to get below the threshold at which your liver and pancreas can function more normally. The fifth group is people willing to change their environment, not merely complete a temporary diet. A 90-day challenge may improve your numbers. But long-term remission requires a system you can continue. Now, who may not be able to reverse diabetes completely? People with type 1 diabetes cannot reverse their disease through weight loss, fasting, supplements, or carbohydrate restriction. Type 1 diabetes is an autoimmune disease in which the immune system destroys the pancreatic beta cells. A person with type 1 diabetes requires insulin. A lower-carbohydrate diet may reduce glucose variability and insulin requirements in some patients, but it does not eliminate the need for insulin. Stopping insulin in type 1 diabetes can cause diabetic ketoacidosis and death. The same warning applies to many people with LADA, or latent autoimmune diabetes in adults. LADA can initially look like type 2 diabetes. The patient may be an adult, may not require insulin immediately, and may temporarily respond to tablets. But the underlying problem is autoimmune beta cell destruction. Tests such as GAD-65, IA-2, ZNT-8 antibodies, and C-peptide may help clarify the diagnosis. If you have LADA, lifestyle still matters tremendously. But the disease cannot simply be fasted away. People with advanced type 2 diabetes may also have a lower chance of full remission. If you have had diabetes for 20 or 30 years, have a very low C-peptide, and require substantial amounts of insulin, much of your beta cell function may have been lost. You can still dramatically improve your glucose. You may reduce your insulin requirements. You may lose weight, improve fatty liver, lower triglycerides, and reduce your risk of complications. But complete medication-free remission may not be realistic. And again, that is not failure. Some people diagnosed with type 2 diabetes actually have other forms of diabetes. Pancreatic surgery, chronic pancreatitis, cystic fibrosis, hemochromatosis, pancreatic cancer, certain medications, Cushing syndrome, acromegaly, and genetic forms of diabetes, can all cause abnormal glucose. There is also monogenic diabetes, including MODY, in which a single genetic abnormality affects glucose regulation. Those conditions require different approaches. If the diagnosis is wrong, the treatment strategy may also be wrong. Now let's build a practical reversal plan. The first target is not glucose. The first target is the cause of the elevated glucose. For many people with type 2 diabetes, that means reducing visceral and ectopic fat, while preserving muscle. Do not simply chase a lower number on the scale. If you lose 30 pounds, but lose a large amount of muscle, you may become lighter without becoming as metabolically healthy as possible. You want to lose fat. You want to preserve, or build, muscle. Remember this phrase, lose the fat, protect the muscle. Let's start with nutrition. There is no single diet that every person with diabetes must follow. Low-carbohydrate diets can work. Mediterranean-style diets can work. Plant-predominant diets can work. Higher-protein diets can work. Structured meal replacement programs can work. Intermittent fasting can work. The common requirement is that the eating pattern must reduce excess energy intake, improve food quality, support adequate protein and nutrition, and be sustainable enough to produce lasting results. The best diet is not the one that creates the most exciting first week. It is the one that creates meaningful improvement you can maintain. Start by removing the most obvious glucose disruptors. Sugary beverages are first. Soda, sweet tea, sweetened coffee, fruit juice, energy drinks, and many smoothies. Deliver glucose quickly without producing enough satiety. Drinking sugar is one of the easiest ways to overwhelm your glucose control system. Water, unsweetened tea, plain coffee, and other non-caloric beverages should become your default choices. Next, reduce refined carbohydrates. White bread, pastries, cookies, candy, sweet cereals, chips, and many packaged snacks are rapidly digested and easy to overeat. That does not mean every person with diabetes must eat zero carbohydrates. It means carbohydrates should earn their place on your plate. Choose foods that bring fiber, nutrients, and satiety, not simply starch and sugar. Beans, lentils, minimally processed whole grains in appropriate portions, vegetables, and whole fruit usually behave differently from flour-based snacks and sweetened drinks. But portions still matter. Healthy carbohydrates can still raise glucose, particularly when the portion is larger than your current metabolic capacity can handle. This is where glucose monitoring becomes educational. Check before a meal, and approximately one to two hours afterward, or use a continuous glucose monitor when appropriate. Do not become frightened by every small rise. Glucose is supposed to increase after eating. Instead, look for patterns. Which meal repeatedly gives you a large, prolonged spike? Was it the bread, the rice, the breakfast cereal, the oversized portion, the lack of protein, or the dessert combined with everything else? Your glucose monitor should be a feedback tool, not a judgment machine. Protein is the next pillar. Adequate protein helps control appetite and preserve muscle during weight loss. Include a meaningful protein source with each major meal. That may include eggs, fish, poultry, Greek yogurt, cottage cheese, lean meat, tofu, tempeh, legumes, or a carefully selected protein supplement. Your exact protein requirement depends on your age, body size, activity level, kidney function, and medical condition. People with advanced kidney disease need individualized guidance, but for many adults trying to lose fat, inadequate protein is a bigger problem than excessive protein. Fiber is also important. Fiber can slow digestion, improve satiety, support bowel health, and reduce the speed of glucose absorption. Vegetables, beans, lentils, seeds, nuts, berries, and appropriate whole food carbohydrates can help. A simple plate structure is this. Half the plate contains non-starchy vegetables. Approximately one quarter contains protein. The remaining portion can contain a high-fiber carbohydrate, additional vegetables, or healthy fat depending on your glucose response and goals. This is not a law. It is a starting framework. Now let's talk about meal timing. If you eat from the moment you wake up until the moment you go to bed, insulin receives very little opportunity to decline. Some people benefit from eliminating unnecessary snacks and creating a consistent overnight fasting window. Start simply. Finish dinner at 7 p.m. and eat breakfast at 7 a.m. That creates a 12-hour overnight fast without doing anything extreme. If you tolerate that well, you may eventually use a 14-hour or 16-hour fasting window. But longer is not automatically better. Intermittent fasting is a tool, not a religion. If fasting causes you to binge later, lose muscle, experience hypoglycemia, or consume inadequate nutrition, it is not helping you. If you take insulin or a sulfonylurea such as glipizide, glimepiride, or glyburide, fasting can cause dangerous hypoglycemia unless your medications are adjusted. Do not combine aggressive fasting with glucose-lowering medication without a plan from your clinician. Now we come to movement. Muscle contractions help move glucose into muscle through pathways that are partly independent of insulin. That means your muscles can absorb more glucose when you use them, even when insulin sensitivity is impaired. One of the simplest interventions is walking after meals. You do not need an exhausting workout. A 10- to 20-minute walk after your largest meal can help reduce the post-meal glucose rise. The message is simple. Do not eat and immediately become furniture. Eat, then move. Resistance training is equally important. Your muscles are your largest glucose disposal system. Building and preserving muscle gives glucose somewhere to go. Perform resistance exercise 2 or 3 times per week, covering the major muscle groups. You can use weights, machines, resistance bands, or body weight exercises. The exact equipment matters less than progressively challenging the muscles safely. Walking improves glucose today. Building muscle improves your capacity to manage glucose tomorrow. You also need aerobic exercise. Brisk walking, cycling, swimming, dancing, or other cardiovascular activities improve fitness, insulin sensitivity, blood pressure, and heart health. A combination of aerobic and resistance training is generally more powerful than relying on either one alone. But remember, exercise cannot consistently outrun excessive food intake. Use nutrition to reduce excess fat. Use resistance training to preserve muscle. Use movement to improve glucose disposal. That is the combination. Sleep is the next pillar, and it is frequently ignored. One night of poor sleep can worsen insulin sensitivity and appetite control the following day. Chronic sleep deprivation can increase hunger, cravings, cortisol, fatigue, and emotional eating. If you snore loudly, stop breathing during sleep, wake with headaches, or remain exhausted despite spending enough time in bed, you may have sleep apnea. Untreated sleep apnea makes glucose and blood pressure more difficult to control. You cannot optimize metabolism while repeatedly suffocating during sleep. Aim for a consistent sleep schedule and approximately seven to nine hours for most adults. Stress also matters. Stress hormones such as cortisol and adrenaline can increase glucose because your body interprets stress as a need for immediate energy. You do not need to eliminate every stressful event. That is impossible. But you need a reliable method of recovery. Walking, prayer, breathing exercises, time outdoors, social connection, therapy, and adequate sleep can all help regulate the stress response. Now let's discuss medication. Some people believe using medication means they failed to reverse diabetes naturally. That belief can be dangerous. Severe hyperglycemia causes glucotoxicity. High glucose itself damages beta cell function and worsens insulin resistance. Sometimes medication or insulin is needed initially to lower glucose quickly and safely. Once glucotoxicity improves and weight decreases, the medication burden may be reduced. Medication can be a bridge to remission. Metformin reduces excessive glucose production by the liver and improves insulin sensitivity. It is inexpensive and widely used. GLP-1 receptor agonists and dual GIP-GLP-1 medications such as semaglutide and terzepatide can substantially reduce appetite, body weight, and glucose. For an appropriate patient, these medications may help produce the fat loss necessary for remission. But they must be combined with adequate protein and resistance training to minimize unnecessary muscle loss. SGLT-2 inhibitors lower glucose by allowing the kidneys to release more glucose into the urine. They can also provide important heart and kidney protection in appropriate patients. Those benefits may justify continuing them even when the A1c looks excellent. This is another reason medication-free remission should not become an obsession. Sometimes the medicine is being used not only for glucose, but to protect the heart or kidneys. Metabolic surgery is another powerful option. For people with significant obesity and type 2 diabetes, procedures such as gastric bypass or sleeve gastrectomy can produce major weight loss and high rates of diabetes improvement or remission. Surgery is not cheating. It changes appetite, gut hormones, food intake, body weight, and metabolism. It also carries risks and requires lifelong nutritional monitoring, so it must be selected carefully. Now let's talk about supplements. I want to be very clear. No supplement reverses diabetes by itself. Supplements do not replace fat loss, muscle building, appropriate nutrition, sleep, monitoring, or prescribed medication. If a supplement lowers your glucose modestly while you continue drinking soda and losing muscle, it has not fixed the disease. Supplements belong at the top of the pyramid, not at the foundation. The foundation is lifestyle and appropriate medical care. Berberine is one of the most studied supplements for metabolic health. It is a plant-derived compound that may influence several pathways involved in glucose and lipid metabolism, including AMPK-related signaling, intestinal glucose handling, the gut microbiome, and liver glucose production. Clinical studies and meta-analyses suggest that berberine can produce modest improvements in fasting glucose, A1c, insulin resistance, triglycerides, and cholesterol in some people. But the studies vary in quality, formulations, doses, and duration. Berberine is not natural ozempic, and it is not a guaranteed substitute for metformin. Traditional berberine also has relatively poor oral absorption and can cause nausea, constipation, diarrhea, cramping, or stomach discomfort. It may interact with medications through drug transporters and liver enzymes. It can also amplify the glucose-lowering effect of diabetes medications. Pregnant or breastfeeding women should generally avoid it, and it should not be given to newborns or infants. SugarMD Super Berberine contains dihydroberberine, also called DHB, along with Ceylon cinnamon. Dihydroberberine is designed to provide a more bioavailable form of berberine in a smaller amount and may be gentler on digestion for some people. Ceylon cinnamon is included to provide additional metabolic and antioxidant support. Super berberine may be considered as an adjunct for an appropriate adult who wants additional glucose support. But the same rules apply. Monitor your glucose. Discuss it with your clinician. Watch for gastrointestinal effects. And do not casually combine it with multiple glucose-lowering products and medications without knowing how your body responds. Gluxion is SugarMD's broader botanical glucose support formula. It includes ingredients such as bitter melon, gymnema, fenugreek, holy basil, turmeric, Indian gooseberry, and other traditional botanical extracts intended to support healthy glucose metabolism. Some individual ingredients have preliminary or modest evidence suggesting effects on glucose absorption, insulin signaling, appetite, oxidative stress, or post-meal glucose. But a multi-ingredient botanical formula should be viewed as supportive, not curative. The more glucose-lowering ingredients you combine, the more carefully you should monitor for low glucose, particularly if you also use insulin, sulfonylureas, or other medications. If you use gluxion or super berberine, use the labeled directions and review your complete medication and supplement list with a knowledgeable clinician or pharmacist. Do not start five new supplements at once. If your glucose improves, or if you develop a side effect, you will not know which product caused it. Introduce supplements thoughtfully. Measure your response. And remember the hierarchy. Food before supplements. Muscle before supplements. Sleep before supplements. Medical safety before supplements. Other nutrients can matter when there is a deficiency. Magnesium deficiency may be associated with insulin resistance. But magnesium supplementation is most rational when intake is inadequate or deficiency is present. Vitamin D deficiency should be corrected for bone and general health. But taking very high doses of vitamin D does not reliably reverse diabetes. Vitamin B12 deserves attention in people using long-term metformin. Because metformin can reduce B12 absorption in some patients. Omega-3 fatty acids can reduce triglycerides. But they should not be advertised as a cure for hyperglycemia. Psyllium and other soluble fibers may help with post-meal glucose, cholesterol, satiety, and bowel regularity. Again, supplements can support a good plan. They cannot rescue a bad plan. Now, how do you know whether your plan is working? Track more than fasting glucose. Fasting glucose tells you what was happening at one moment. A 1C estimates your average glucose over roughly 2-3 months. Although anemia, kidney disease, hemoglobin variance, blood loss, transfusion, and other factors can affect its accuracy. Post-meal glucose tells you how your body handles a specific meal. A continuous glucose monitor can reveal patterns that finger stick testing may miss. But do not become obsessed with producing a perfectly flat line. A normal pancreas does not create perfectly flat glucose. The goal is improved control, not glucose anxiety. Track your waist circumference because visceral fat matters. Track your body composition when possible. Track your strength because muscle matters. Track your blood pressure, triglycerides, HDL, liver enzymes, kidney function, urine albumin, and eye examinations. Diabetes is not only a glucose disease. It is a cardiovascular, kidney, nerve, eye, liver, and metabolic disease. Even if you achieve remission, continue medical follow-up. Prior exposure to diabetes may still carry risk. You still need periodic A1C testing. Blood pressure management. Cholesterol assessment. Kidney screening. Foot care. And eye examinations based on your clinical situation. Remission does not erase your history. Now let me give you a practical starting plan. First, confirm what type of diabetes you actually have. Second, determine whether you still have meaningful insulin production when clinically appropriate. Third, identify your biggest drivers. Excess visceral fat. Poor food quality. Inactivity. Sleep apnea. Medication effects. Severe stress. Or another endocrine disorder. Fourth, eliminate sugary beverages. Fifth, build meals around protein, vegetables, fiber, and minimally processed foods. Sixth, reduce refined carbohydrates. And adjust total carbohydrate intake based on your glucose response. Seventh, create a sustainable calorie deficit if you have excess body fat. Eighth, stop unnecessary grazing. And consider a reasonable overnight fasting window if it is safe for you. Ninth, walk after meals. Tenth, perform resistance training two or three times per week. Eleventh, protect your sleep. And evaluate possible sleep apnea. Twelfth, use medications strategically rather than viewing them as defeat. Thirteenth, consider carefully selected supplements only after the foundation is in place. And finally, measure the result. Do not guess. Check the glucose. Check the A1C. Check the waist. Check the strength. Check the body composition. Then adjust the plan. Can type 2 diabetes be reversed? For many people, yes. Especially when it is identified early. Insulin production remains adequate. And enough excess fat can be lost and kept off. For others, complete remission may not be possible. But improvement is almost always valuable. Reducing an A1C from 10% to 7% matters. Reducing insulin from 100 units per day to 30 units matters. Losing visceral fat matters. Building muscle matters. Improving fatty liver matters. Preventing kidney failure matters. You do not need a perfect outcome to achieve a life-changing outcome. The biggest mistake is waiting. People spend years asking whether they should try low carb, Mediterranean, fasting, metformin, GLP, one therapy, resistance training, or supplements. Meanwhile, the beta cells remain under pressure. Do not wait for the perfect plan. Start with the fundamentals. Remove liquid sugar. Eat real food. Prioritize protein and fiber. Lose excess fat. Protect your muscle. Move after meals. Sleep properly. Take necessary medication. Use supplements intelligently, not emotionally. And monitor the results. Here is the final message I want you to remember. Type 2 diabetes is not simply too much sugar in the blood. It is a failure of the entire energy management system. The liver releases too much glucose. The muscles do not absorb glucose efficiently. The fat tissue exceeds its safe storage capacity. The pancreas works overtime and eventually struggles to keep up. To reverse the disease process, you must reverse as many of those abnormalities as possible. Reduce liver and visceral fat. Improve muscle insulin sensitivity. Give glucose somewhere to go. Reduce the burden on the pancreas. And intervene before too much beta cell function is lost. That is how you move beyond controlling a number. That is how you begin treating the disease at its roots. Remission is not guaranteed. It is not permanent unless the metabolic improvements are maintained. And it is not a reason to stop medical monitoring. But it is real. It is possible. And for the right person, with the right plan, started at the right time, it can completely change the future. Do not simply manage your glucose. Understand why it is elevated. Do not simply lose weight. Lose visceral fat and preserve muscle. Do not simply collect supplements. Build the foundation first. And do not let anybody tell you that type 2 diabetes must always move in only one direction. For many people, the process can be interrupted. It can be improved. And sometimes it can be pushed into remission. Good luck. And I'll see you in the next video.